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Water-soluble artemisinin derivatives as promising therapeutic immunosuppressants of autoimmune diseases.

Cellular & molecular immunology

Li H, Zuo J, Tang W.
PMID: 28890548
Cell Mol Immunol. 2017 Sep 11; doi: 10.1038/cmi.2017.87. Epub 2017 Sep 11.

No abstract available.

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Li H, Zuo J, Tang W. Water-soluble artemisinin derivatives as promising therapeutic immunosuppressants of autoimmune diseases. Cell Mol Immunol. 2017;doi: 10.1038/cmi.2017.87.
Li, H., Zuo, J., & Tang, W. (2017). Water-soluble artemisinin derivatives as promising therapeutic immunosuppressants of autoimmune diseases. Cellular & molecular immunology, . https://doi.org/10.1038/cmi.2017.87
Li, Heng, et al. "Water-soluble artemisinin derivatives as promising therapeutic immunosuppressants of autoimmune diseases." Cellular & molecular immunology vol. (2017). doi: https://doi.org/10.1038/cmi.2017.87
Li H, Zuo J, Tang W. Water-soluble artemisinin derivatives as promising therapeutic immunosuppressants of autoimmune diseases. Cell Mol Immunol. 2017 Sep 11; doi: 10.1038/cmi.2017.87. Epub 2017 Sep 11. PMID: 28890548; PMCID: PMC5675962.
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Role of programmed cell death 4 in diseases: a double-edged sword.

Cellular & molecular immunology

Jiang Y, Jia Y, Zhang L.
PMID: 28920585
Cell Mol Immunol. 2017 Sep 18; doi: 10.1038/cmi.2017.84. Epub 2017 Sep 18.

No abstract available.

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Jiang Y, Jia Y, Zhang L. Role of programmed cell death 4 in diseases: a double-edged sword. Cell Mol Immunol. 2017;doi: 10.1038/cmi.2017.84.
Jiang, Y., Jia, Y., & Zhang, L. (2017). Role of programmed cell death 4 in diseases: a double-edged sword. Cellular & molecular immunology, . https://doi.org/10.1038/cmi.2017.84
Jiang, Yang, et al. "Role of programmed cell death 4 in diseases: a double-edged sword." Cellular & molecular immunology vol. (2017). doi: https://doi.org/10.1038/cmi.2017.84
Jiang Y, Jia Y, Zhang L. Role of programmed cell death 4 in diseases: a double-edged sword. Cell Mol Immunol. 2017 Sep 18; doi: 10.1038/cmi.2017.84. Epub 2017 Sep 18. PMID: 28920585; PMCID: PMC5675960.
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Complement-endothelial cell interactions: pathophysiological implications.

Molecular immunology

Tedesco F, Fischetti F, Pausa M, Dobrina A, Sim RB, Daha MR.
PMID: 10781839
Mol Immunol. 2000 Jan;37(1):91. doi: 10.1016/s0161-5890(00)00036-5.

No abstract available.

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Tedesco F, Fischetti F, Pausa M, et al. Complement-endothelial cell interactions: pathophysiological implications. Mol Immunol. 2000;37(1):91doi: 10.1016/s0161-5890(00)00036-5.
Tedesco, F., Fischetti, F., Pausa, M., Dobrina, A., Sim, R. B., & Daha, M. R. (2000). Complement-endothelial cell interactions: pathophysiological implications. Molecular immunology, 37(1), 91. https://doi.org/10.1016/s0161-5890(00)00036-5
Tedesco, et al. "Complement-endothelial cell interactions: pathophysiological implications." Molecular immunology vol. 37,1 (2000): 91. doi: https://doi.org/10.1016/s0161-5890(00)00036-5
Tedesco F, Fischetti F, Pausa M, Dobrina A, Sim RB, Daha MR. Complement-endothelial cell interactions: pathophysiological implications. Mol Immunol. 2000 Jan;37(1):91. doi: 10.1016/s0161-5890(00)00036-5. PMID: 10781839.
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IL-17 initiates tertiary lymphoid organ formation.

Cellular & molecular immunology

Zhang X, Lu B.
PMID: 22179673
Cell Mol Immunol. 2012 Jan;9(1):9-10. doi: 10.1038/cmi.2011.48. Epub 2011 Dec 19.

No abstract available.

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Zhang X, Lu B. IL-17 initiates tertiary lymphoid organ formation. Cell Mol Immunol. 2011;9(1):9-10doi: 10.1038/cmi.2011.48.
Zhang, X., & Lu, B. (2012). IL-17 initiates tertiary lymphoid organ formation. Cellular & molecular immunology, 9(1), 9-10. https://doi.org/10.1038/cmi.2011.48
Zhang, Xueguang, and Lu, Binfeng. "IL-17 initiates tertiary lymphoid organ formation." Cellular & molecular immunology vol. 9,1 (2012): 9-10. doi: https://doi.org/10.1038/cmi.2011.48
Zhang X, Lu B. IL-17 initiates tertiary lymphoid organ formation. Cell Mol Immunol. 2012 Jan;9(1):9-10. doi: 10.1038/cmi.2011.48. Epub 2011 Dec 19. PMID: 22179673; PMCID: PMC4002935.
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Analyzing the phenotypic and functional complexity of lymphocytes using CyTOF (cytometry by time-of-flight).

Cellular & molecular immunology

Chen G, Weng NP.
PMID: 22635255
Cell Mol Immunol. 2012 Jul;9(4):322-3. doi: 10.1038/cmi.2012.16. Epub 2012 May 28.

No abstract available.

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Chen G, Weng NP. Analyzing the phenotypic and functional complexity of lymphocytes using CyTOF (cytometry by time-of-flight). Cell Mol Immunol. 2012;9(4):322-3doi: 10.1038/cmi.2012.16.
Chen, G., & Weng, N. P. (2012). Analyzing the phenotypic and functional complexity of lymphocytes using CyTOF (cytometry by time-of-flight). Cellular & molecular immunology, 9(4), 322-3. https://doi.org/10.1038/cmi.2012.16
Chen, Guobing, and Weng, Nan-ping. "Analyzing the phenotypic and functional complexity of lymphocytes using CyTOF (cytometry by time-of-flight)." Cellular & molecular immunology vol. 9,4 (2012): 322-3. doi: https://doi.org/10.1038/cmi.2012.16
Chen G, Weng NP. Analyzing the phenotypic and functional complexity of lymphocytes using CyTOF (cytometry by time-of-flight). Cell Mol Immunol. 2012 Jul;9(4):322-3. doi: 10.1038/cmi.2012.16. Epub 2012 May 28. PMID: 22635255; PMCID: PMC4012870.
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Correction to: TAOK1 negatively regulates IL-17-mediated signaling and inflammation.

Cellular & molecular immunology

Zhang Z, Tang Z, Ma X, Sun K, Fan L, Fang J, Pan J, Wang X, An H, Zhou J.
PMID: 29844589
Cell Mol Immunol. 2018 Oct;15(10):940. doi: 10.1038/s41423-018-0048-5.

In this article, published online 5 February 2018, the second primer of mouse actin, il-6, il-1β, TNFα, cxcl1, cxcl2, and ccl20 in Table 2 should be marked as "reverse primer" instead of "forward primer". Corrected Table 2 is shown...

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Zhang Z, Tang Z, Ma X, et al. Correction to: TAOK1 negatively regulates IL-17-mediated signaling and inflammation. Cell Mol Immunol. 2018;15(10):940doi: 10.1038/s41423-018-0048-5.
Zhang, Z., Tang, Z., Ma, X., Sun, K., Fan, L., Fang, J., Pan, J., Wang, X., An, H., & Zhou, J. (2018). Correction to: TAOK1 negatively regulates IL-17-mediated signaling and inflammation. Cellular & molecular immunology, 15(10), 940. https://doi.org/10.1038/s41423-018-0048-5
Zhang, Zhaoru, et al. "Correction to: TAOK1 negatively regulates IL-17-mediated signaling and inflammation." Cellular & molecular immunology vol. 15,10 (2018): 940. doi: https://doi.org/10.1038/s41423-018-0048-5
Zhang Z, Tang Z, Ma X, Sun K, Fan L, Fang J, Pan J, Wang X, An H, Zhou J. Correction to: TAOK1 negatively regulates IL-17-mediated signaling and inflammation. Cell Mol Immunol. 2018 Oct;15(10):940. doi: 10.1038/s41423-018-0048-5. PMID: 29844589; PMCID: PMC6207561.
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Suppression of monosodium urate crystal-induced inflammation by inhibiting TGF-β-activated kinase 1-dependent signaling: role of the ubiquitin proteasome system.

Cellular & molecular immunology

Singh AK, Haque M, O'Sullivan K, Chourasia M, Ouseph MM, Ahmed S.
PMID: 31511642
Cell Mol Immunol. 2021 Jan;18(1):162-170. doi: 10.1038/s41423-019-0284-3. Epub 2019 Sep 11.

Monosodium urate (MSU) crystals activate inflammatory pathways that overlap with interleukin-1β (IL-1β) signaling. However, the post-translational mechanisms involved and the role of signaling proteins in this activation are unknown. In the present study, we investigated the intracellular signaling mechanisms...

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Singh AK, Haque M, O'Sullivan K, et al. Suppression of monosodium urate crystal-induced inflammation by inhibiting TGF-β-activated kinase 1-dependent signaling: role of the ubiquitin proteasome system. Cell Mol Immunol. 2019;18(1):162-170doi: 10.1038/s41423-019-0284-3.
Singh, A. K., Haque, M., O'Sullivan, K., Chourasia, M., Ouseph, M. M., & Ahmed, S. (2021). Suppression of monosodium urate crystal-induced inflammation by inhibiting TGF-β-activated kinase 1-dependent signaling: role of the ubiquitin proteasome system. Cellular & molecular immunology, 18(1), 162-170. https://doi.org/10.1038/s41423-019-0284-3
Singh, Anil K, et al. "Suppression of monosodium urate crystal-induced inflammation by inhibiting TGF-β-activated kinase 1-dependent signaling: role of the ubiquitin proteasome system." Cellular & molecular immunology vol. 18,1 (2021): 162-170. doi: https://doi.org/10.1038/s41423-019-0284-3
Singh AK, Haque M, O'Sullivan K, Chourasia M, Ouseph MM, Ahmed S. Suppression of monosodium urate crystal-induced inflammation by inhibiting TGF-β-activated kinase 1-dependent signaling: role of the ubiquitin proteasome system. Cell Mol Immunol. 2021 Jan;18(1):162-170. doi: 10.1038/s41423-019-0284-3. Epub 2019 Sep 11. PMID: 31511642; PMCID: PMC7853128.
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Correction to: Adenovirus-mediated LIGHT gene modification in murine B-cell lymphoma elicits a potent antitumor effect.

Cellular & molecular immunology

Hu G, Liu Y, Li H, Zhao D, Yang L, Shen J, Hong X, Cao X, Wang Q.
PMID: 32071419
Cell Mol Immunol. 2020 Mar;17(3):305-306. doi: 10.1038/s41423-019-0356-4.

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

Cite
Hu G, Liu Y, Li H, et al. Correction to: Adenovirus-mediated LIGHT gene modification in murine B-cell lymphoma elicits a potent antitumor effect. Cell Mol Immunol. 2020;17(3):305-306doi: 10.1038/s41423-019-0356-4.
Hu, G., Liu, Y., Li, H., Zhao, D., Yang, L., Shen, J., Hong, X., Cao, X., & Wang, Q. (2020). Correction to: Adenovirus-mediated LIGHT gene modification in murine B-cell lymphoma elicits a potent antitumor effect. Cellular & molecular immunology, 17(3), 305-306. https://doi.org/10.1038/s41423-019-0356-4
Hu, Guili, et al. "Correction to: Adenovirus-mediated LIGHT gene modification in murine B-cell lymphoma elicits a potent antitumor effect." Cellular & molecular immunology vol. 17,3 (2020): 305-306. doi: https://doi.org/10.1038/s41423-019-0356-4
Hu G, Liu Y, Li H, Zhao D, Yang L, Shen J, Hong X, Cao X, Wang Q. Correction to: Adenovirus-mediated LIGHT gene modification in murine B-cell lymphoma elicits a potent antitumor effect. Cell Mol Immunol. 2020 Mar;17(3):305-306. doi: 10.1038/s41423-019-0356-4. PMID: 32071419; PMCID: PMC7051978.
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Conversion of effector CD4.

Cellular & molecular immunology

Robins E, Zheng M, Ni Q, Liu S, Liang C, Zhang B, Guo J, Zhuang Y, He YW, Zhu P, Wan Y, Li QJ.
PMID: 32066854
Cell Mol Immunol. 2021 Jan;18(1):150-161. doi: 10.1038/s41423-019-0347-5. Epub 2020 Feb 17.

CD4

Cite
Robins E, Zheng M, Ni Q, et al. Conversion of effector CD4. Cell Mol Immunol. 2020;18(1):150-161doi: 10.1038/s41423-019-0347-5.
Robins, E., Zheng, M., Ni, Q., Liu, S., Liang, C., Zhang, B., Guo, J., Zhuang, Y., He, Y. W., Zhu, P., Wan, Y., & Li, Q. J. (2021). Conversion of effector CD4. Cellular & molecular immunology, 18(1), 150-161. https://doi.org/10.1038/s41423-019-0347-5
Robins, Elizabeth, et al. "Conversion of effector CD4." Cellular & molecular immunology vol. 18,1 (2021): 150-161. doi: https://doi.org/10.1038/s41423-019-0347-5
Robins E, Zheng M, Ni Q, Liu S, Liang C, Zhang B, Guo J, Zhuang Y, He YW, Zhu P, Wan Y, Li QJ. Conversion of effector CD4. Cell Mol Immunol. 2021 Jan;18(1):150-161. doi: 10.1038/s41423-019-0347-5. Epub 2020 Feb 17. PMID: 32066854; PMCID: PMC7853072.
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Corrigendum to "Nicorandil modulated macrophages activation and polarization via NF-κb signaling pathway" [Mol. Immunol. 88 (2017) 69-78].

Molecular immunology

Zhang F, Xuan Y, Cui J, Liu X, Shao Z, Yu B.
PMID: 32805610
Mol Immunol. 2020 Oct;126:110. doi: 10.1016/j.molimm.2020.07.010. Epub 2020 Aug 14.

No abstract available.

Cite
Zhang F, Xuan Y, Cui J, et al. Corrigendum to "Nicorandil modulated macrophages activation and polarization via NF-κb signaling pathway" [Mol. Immunol. 88 (2017) 69-78]. Mol Immunol. 2020;126:110doi: 10.1016/j.molimm.2020.07.010.
Zhang, F., Xuan, Y., Cui, J., Liu, X., Shao, Z., & Yu, B. (2020). Corrigendum to "Nicorandil modulated macrophages activation and polarization via NF-κb signaling pathway" [Mol. Immunol. 88 (2017) 69-78]. Molecular immunology, 126110. https://doi.org/10.1016/j.molimm.2020.07.010
Zhang, Fengyun, et al. "Corrigendum to "Nicorandil modulated macrophages activation and polarization via NF-κb signaling pathway" [Mol. Immunol. 88 (2017) 69-78]." Molecular immunology vol. 126 (2020): 110. doi: https://doi.org/10.1016/j.molimm.2020.07.010
Zhang F, Xuan Y, Cui J, Liu X, Shao Z, Yu B. Corrigendum to "Nicorandil modulated macrophages activation and polarization via NF-κb signaling pathway" [Mol. Immunol. 88 (2017) 69-78]. Mol Immunol. 2020 Oct;126:110. doi: 10.1016/j.molimm.2020.07.010. Epub 2020 Aug 14. PMID: 32805610.
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Correction to: T follicular helper expansion and humoral-mediated rejection are independent of the HVEM/BTLA pathway.

Cellular & molecular immunology

Rodriguez-Barbosa JI, Fernandez-Renedo C, Moral AMB, Bühler L, Del Rio ML.
PMID: 30635646
Cell Mol Immunol. 2019 Mar;16(3):314. doi: 10.1038/s41423-018-0196-7.

In this article, one of the grating agencies requested us to incorporate the information, Spanish Government and co-funded by European Union ERDF/ESF, "Investing in your future", in the acknowledgments section. The correct acknowledgement is as follows: "This work has...

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Rodriguez-Barbosa JI, Fernandez-Renedo C, Moral AMB, et al. Correction to: T follicular helper expansion and humoral-mediated rejection are independent of the HVEM/BTLA pathway. Cell Mol Immunol. 2019;16(3):314doi: 10.1038/s41423-018-0196-7.
Rodriguez-Barbosa, J. I., Fernandez-Renedo, C., Moral, A. M. B., Bühler, L., & Del Rio, M. L. (2019). Correction to: T follicular helper expansion and humoral-mediated rejection are independent of the HVEM/BTLA pathway. Cellular & molecular immunology, 16(3), 314. https://doi.org/10.1038/s41423-018-0196-7
Rodriguez-Barbosa, Jose-Ignacio, et al. "Correction to: T follicular helper expansion and humoral-mediated rejection are independent of the HVEM/BTLA pathway." Cellular & molecular immunology vol. 16,3 (2019): 314. doi: https://doi.org/10.1038/s41423-018-0196-7
Rodriguez-Barbosa JI, Fernandez-Renedo C, Moral AMB, Bühler L, Del Rio ML. Correction to: T follicular helper expansion and humoral-mediated rejection are independent of the HVEM/BTLA pathway. Cell Mol Immunol. 2019 Mar;16(3):314. doi: 10.1038/s41423-018-0196-7. PMID: 30635646; PMCID: PMC6460492.
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Sox12 enhances Fbw7-mediated ubiquitination and degradation of GATA3 in Th2 cells.

Cellular & molecular immunology

Suehiro KI, Suto A, Suga K, Furuya H, Iwata A, Iwamoto T, Tanaka S, Kageyama T, Suzuki K, Hirose K, Lefebvre V, Nakajima H.
PMID: 32152552
Cell Mol Immunol. 2021 Jul;18(7):1729-1738. doi: 10.1038/s41423-020-0384-0. Epub 2020 Mar 09.

Allergic asthma that is caused by inhalation of house dust mites (HDMs) is mainly mediated by Th2 cells. Recently, the roles of Sox (SRY-related high-mobility-group (HMG)-box) family members in various immune responses have been investigated. However, the roles of...

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Suehiro KI, Suto A, Suga K, et al. Sox12 enhances Fbw7-mediated ubiquitination and degradation of GATA3 in Th2 cells. Cell Mol Immunol. 2020;18(7):1729-1738doi: 10.1038/s41423-020-0384-0.
Suehiro, K. I., Suto, A., Suga, K., Furuya, H., Iwata, A., Iwamoto, T., Tanaka, S., Kageyama, T., Suzuki, K., Hirose, K., Lefebvre, V., & Nakajima, H. (2021). Sox12 enhances Fbw7-mediated ubiquitination and degradation of GATA3 in Th2 cells. Cellular & molecular immunology, 18(7), 1729-1738. https://doi.org/10.1038/s41423-020-0384-0
Suehiro, Ken-Ichi, et al. "Sox12 enhances Fbw7-mediated ubiquitination and degradation of GATA3 in Th2 cells." Cellular & molecular immunology vol. 18,7 (2021): 1729-1738. doi: https://doi.org/10.1038/s41423-020-0384-0
Suehiro KI, Suto A, Suga K, Furuya H, Iwata A, Iwamoto T, Tanaka S, Kageyama T, Suzuki K, Hirose K, Lefebvre V, Nakajima H. Sox12 enhances Fbw7-mediated ubiquitination and degradation of GATA3 in Th2 cells. Cell Mol Immunol. 2021 Jul;18(7):1729-1738. doi: 10.1038/s41423-020-0384-0. Epub 2020 Mar 09. PMID: 32152552; PMCID: PMC8245422.
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Showing 1 to 12 of 1010 entries